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Robert P Friedland, MD
Formerly, Professor of Neurology
Department of Pathology
School of Medicine
Case Western Reserve University
Dr Friedland is a clinical and research neurologist interested in behavior and aging. He is a graduate of the City College of New York and the Mount Sinai School of Medicine (1973). He completed his neurology residency at the Mount Sinai Hospital in New York and from 1977-1978 was a Fellow in dementia and aging under Dr. Robert Katzman at the Albert Einstein College of Medicine, NY. Since then he has worked at the University of California Davis and the Research Medicine Group of the Lawrence Berkeley Laboratory of the University California, Berkeley. He served from 1985 to 1990 as Deputy Clinical Director and Chief of the Section on Brain Aging and Dementia of the National Institute on Aging Institute, National Institutes of Health, Bethesda, MD. Since 1990 he has worked at Case Western Reserve University in the School of Medicine, where he is currently Professor of Neurology and Psychiatry and Chief of the Laboratory of Neurogeriatrics. Dr. Friedland’s work has focused on clinical and biological issues in Alzheimer’s disease and related disorders. He was among the first to document patterns of abnormal brain metabolism and cerebral blood flow in the disease using positron emission and single photon emission computed tomography. He also contributed early studies on the use of magnetic resonance imaging in dementia. The first studies of problems of driving behavior in patients with Alzheimer’s disease came from his group at the NIH. More recently, Dr. Friedland has been working on the patterns of disease occurrence and risk and protective factors, with studies of the Kikuku in Kenya, Jews and Arabs in Israel and Caucasian and African-American subjects in Cleveland. He has documented a series of important determinants of the disease, including physical and mental inactivity, smoking and diet. This work has focused on interactions of genetic and environmental lifestyle elements. His group is also using animal models to better define the pathophysiology of Alzheimer’s disease and develop new treatments.